volume 16 issue 4 pages 633-642

Depression and Inflammatory Bowel Disease: A Bidirectional Two-sample Mendelian Randomization Study

Publication typeJournal Article
Publication date2021-11-05
scimago Q1
wos Q1
SJR2.991
CiteScore15.6
Impact factor8.7
ISSN18739946, 18764479
General Medicine
Gastroenterology
Abstract
Background and Aims

Observational studies have suggested a bidirectional association between depression and inflammatory bowel disease [IBD], including Crohn’s disease [CD] and ulcerative colitis [UC]. However, it remains unclear whether the observed associations are causal due to the difficulties of determining sequential temporality. We investigated the association between depression and IBD by using bidirectional two-sample Mendelian randomization [MR].

Methods

Independent genetic variants for depression and IBD were selected as instruments from published genome-wide association studies [GWAS] among individuals of predominantly European ancestry. Summary statistics for instrument–outcome associations were retrieved from three separate databases for both depression [Psychiatric Genomics Consortium, FinnGen and UK Biobank] and IBD [the largest GWAS meta-analysis, FinnGen and UK Biobank], respectively. MR analyses included the inverse-variance-weighted method, weighted-median estimator, MR-Egger regression, and sensitivity analyses of Steiger filtering and MR PRESSO. From either direction, analyses were performed per outcome database and were subsequently meta-analysed using a fixed-effect model.

Results

Genetically predicted depression [per log-odds ratio increase] was associated with a higher risk of IBD; odds ratios [95% confidence interval] for IBD, CD and UC were 1.20 [1.05, 1.36], 1.29 [1.07, 1.56] and 1.22 [1.01, 1.47] in a combined sample size of 693 183 [36 507 IBD cases], 212 172 [13 714 CD cases] and 219 686 [15 691 UC cases] individuals, respectively. In contrast, no association was observed between genetically influenced IBD and depression in 534 635 individuals [71 466 depression cases].

Conclusions

Our findings corroborated a causal association of depression on IBD, which may impact the clinical decision on the management of depression in patients with IBD. Though our results did not support a causal effect of IBD on depression, further investigations are needed to clarify the effect of IBD activity on depression [with different symptomology].

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GOST |
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GOST Copy
Luo J. et al. Depression and Inflammatory Bowel Disease: A Bidirectional Two-sample Mendelian Randomization Study // Journal of Crohn's and Colitis. 2021. Vol. 16. No. 4. pp. 633-642.
GOST all authors (up to 50) Copy
Luo J., Xu Z., Noordam R., van Heemst D., Li-Gao R. Depression and Inflammatory Bowel Disease: A Bidirectional Two-sample Mendelian Randomization Study // Journal of Crohn's and Colitis. 2021. Vol. 16. No. 4. pp. 633-642.
RIS |
Cite this
RIS Copy
TY - JOUR
DO - 10.1093/ecco-jcc/jjab191
UR - https://doi.org/10.1093/ecco-jcc/jjab191
TI - Depression and Inflammatory Bowel Disease: A Bidirectional Two-sample Mendelian Randomization Study
T2 - Journal of Crohn's and Colitis
AU - Luo, Jiao
AU - Xu, Zhongwei
AU - Noordam, Raymond
AU - van Heemst, Diana
AU - Li-Gao, Ruifang
PY - 2021
DA - 2021/11/05
PB - Oxford University Press
SP - 633-642
IS - 4
VL - 16
PMID - 34739073
SN - 1873-9946
SN - 1876-4479
ER -
BibTex |
Cite this
BibTex (up to 50 authors) Copy
@article{2021_Luo,
author = {Jiao Luo and Zhongwei Xu and Raymond Noordam and Diana van Heemst and Ruifang Li-Gao},
title = {Depression and Inflammatory Bowel Disease: A Bidirectional Two-sample Mendelian Randomization Study},
journal = {Journal of Crohn's and Colitis},
year = {2021},
volume = {16},
publisher = {Oxford University Press},
month = {nov},
url = {https://doi.org/10.1093/ecco-jcc/jjab191},
number = {4},
pages = {633--642},
doi = {10.1093/ecco-jcc/jjab191}
}
MLA
Cite this
MLA Copy
Luo, Jiao, et al. “Depression and Inflammatory Bowel Disease: A Bidirectional Two-sample Mendelian Randomization Study.” Journal of Crohn's and Colitis, vol. 16, no. 4, Nov. 2021, pp. 633-642. https://doi.org/10.1093/ecco-jcc/jjab191.