Current Treatment Options in Psychiatry
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SCImago
Q1
SJR
1.054
CiteScore
5.7
Categories
Clinical Psychology
Psychiatry and Mental Health
Areas
Medicine
Psychology
Years of issue
2014-2025
journal names
Current Treatment Options in Psychiatry
Top-3 citing journals

Frontiers in Psychiatry
(96 citations)

Current Treatment Options in Psychiatry
(53 citations)

Journal of Affective Disorders
(41 citations)
Top-3 organizations

Harvard University
(28 publications)

Boston University
(19 publications)

Icahn School of Medicine at Mount Sinai
(16 publications)

Harvard University
(15 publications)

Boston University
(10 publications)

Stanford University
(8 publications)
Most cited in 5 years
Found
Publications found: 8233
Q1

Central Executive Network drives delta-9-tetrahydrocannabinol (THC)-induced nonlinear changes in large-scale functional connectivity in adolescent nonhuman primates
Byun A.J., Deshpande H.U., Stover J., Kangas B.D., Kohut S.J.
Q1
Neuropsychopharmacology
,
2025
,
citations by CoLab: 0

Q1

A new module in the drug development process: preclinical multi-center randomized controlled trial of R-ketamine on alcohol relapse
Meinhardt M.W., Skorodumov I., Jeanblanc J., Benvenuti F., Hilal F.F., Domi E., André C., Bodeau S., Jeanblanc V., Domanegg K., Ciccocioppo R., Naassila M., Spanagel R.
Abstract
The drug development process in psychiatry faces significant challenges due to low reproducibility rates in animal testing, which often leads to translation failures. To address this issue, we introduce a new approach in psychiatric drug development: a preclinical randomized controlled trial (preRCT). To demonstrate its potential utility, we conducted a multi-center preRCT using the alcohol deprivation effect (ADE) model to assess the impact of ketamine and R-ketamine on alcohol relapse across three European research centers. Ketamine (20 mg/kg) significantly reduced relapse, while R-ketamine showed efficacy only in females. A higher dose of R-ketamine (40 mg/kg) was also effective in males. These sex-dependent effects were linked to plasma R-ketamine levels, which were two-fold higher in female compared to male rats. Notably, R-ketamine demonstrated a lasting reduction in alcohol consumption without adverse effects. In conclusion, our preRCT demonstrates R-ketamine’s effectiveness in reducing alcohol relapse and supports translation to a clinical RCT that accounts for sex-dependent effects.
Q1

The crosstalk between CREB and PER2 mediates the transition between mania- and depression-like behavior
Wang X., Ji Y., Li S., Serchov T.
Abstract
Bipolar disorder (BD) is a severe psychiatric disorder characterized by alternating manic and depressive episodes. The molecular mechanisms underlying the transition between mania and depression remain unclear. Utilizing a mania animal model induced by ouabain, we observed reduced phosphorylated level of cyclic AMP-responsive element-binding protein (pCREB) and Period (PER)2 expression in the cornu ammonis (CA1) region of the hippocampus, which were restored by lithium treatment. shRNA knockdown of CREB or Per2 in CA1 region induced mania-like behavior, while overexpression of both factors resulted in depression-like behavior. Furthermore, our protein analyses revealed that the upregulation or downregulation of CREB or Per2 influenced each other’s expression. Co-immunoprecipitation results demonstrated that CREB interacts with PER2. Taken together, our data suggest for potential inter-regulatory crosstalk between CREB–PER2 in hippocampal CA1 region, which mediates the transition between mania- and depression-like behaviors.
Q1

Pharmacological reduction of reverse-translated hippocampal hyperactivity in mouse: relevance for psychosis
Dybowski F.P., Scott D.S., Tamminga C.A.
Q1
Neuropsychopharmacology
,
2025
,
citations by CoLab: 0

Q1

Longitudinal associations between white matter integrity, early life adversities, and treatment response following cognitive-behavioral therapy in depression
Flinkenflügel K., Borgers T., Klug M., Mummendey M.M., Leehr E.J., Meinert S., Gruber M., Repple J., Kircher T., Opel N., Bauer J., Zwiky E., König P., Küttner A., Schöniger K., et. al.
Abstract
Cognitive-behavioral therapy (CBT) is a primary treatment for depression. Although previous research has underscored the significant roles of white matter (WM) alterations and maladaptive parenting in depression risk, their associations with CBT response remain largely unknown. This longitudinal study investigated the interplay of WM integrity changes over time, treatment response, and parenting style in patients with depression. Diffusion-tensor-imaging and clinical data were assessed in n = 65 (55% female) patients with depression before and after 20 CBT sessions and n = 65 (68% female) healthy controls (HC) in a naturalistic design. Linear-mixed-effect models compared changes in fractional anisotropy (FA) between groups and tested associations between FA changes and symptom changes. It was investigated whether parenting style predicts depressive symptoms at follow-up and whether FA changes mediate this association. Patients showed differential FA changes over time in the corpus callosum and corona radiata compared to HC (p
tfce-FWE = 0.008). Increases in FA in the corpus callosum, corona radiata and superior longitudinal fasciculus were linked to symptom improvement after CBT in patients (p
tfce-FWE = 0.023). High parental care (p
FDR = 0.010) and low maternal overprotection (p
FDR = 0.001) predicted fewer depressive symptoms at follow-up. The association between maternal overprotection and depressive symptoms at follow-up was mediated by FA changes (p
FDR = 0.044). Robustness checks—controlling for outliers, non-linear age effects, clinical characteristics, and patient subgroups—supported these results. Overall, patients with depression show changes in WM integrity following CBT, which are linked to treatment response. The results highlight the significance of early life adversities and related microstructural changes in the effectiveness of CBT for treating depression.
Q1

Protein interacting with C-kinase 1 (PICK1) regulates synaptic function and reversal learning in a mouse model for schizophrenia
Samsom J.N., Xu M., Ávila A., Daskalakis A.A., Dai J.H., Gao X., Georgiou J., Collingridge G.L., Liu F., Wong A.H.
Q1
Neuropsychopharmacology
,
2025
,
citations by CoLab: 0

Q1

Prediction of alcohol intake patterns with olfactory and gustatory brain connectivity networks
Agarwal K., Chaudhary S., Tomasi D., Volkow N.D., Joseph P.V.
Abstract
Craving in alcohol drinkers is often triggered by chemosensory cues, such as taste and smell, which are linked to brain network connectivity. This study aimed to investigate whether these brain connectivity patterns could predict alcohol intake in young adults. Resting-state fMRI data were obtained from the Human Connectome Project (HCP) Young Adult cohort, comprising 1003 participants. Functional connectomes generated from 100 independent components were analyzed, identifying significant connections correlated with taste and odor scores after applying a false discovery rate (FDR) correction using the Benjamini-Hochberg (BH) method. These significant connections were then utilized as predictors in general linear models for various alcohol intake metrics. The models were validated in an independent sample to assess their accuracy. The training sample (n = 702) and the validation sample (n = 117) showed no significant demographic differences. Out of 742 possible connections, 41 related to odor and 25 related to taste passed the significance threshold (P < 0.05) after FDR-BH correction. Notable predictors included visual-visual connectivity (node32-node13: β = 0.028, P = 0.02) for wine consumption and connectivity between the ventral attention network (VAN) and the frontal parietal/caudate nucleus (FP/CN) (node27-node9: β = −0.31, P = 0.04) for total alcohol intake in the past-week and maximum number of drinks per day in the past-year. The predictive models demonstrated strong accuracy, with root mean square error (RMSE) values of 5.15 for odor-related models and 5.14 for taste-related models. The F1 scores were 0.74 for the odor model and 0.71 for the taste model, indicating reliable performance. These findings suggest that specific patterns of brain connectivity associated with taste and olfactory perception may serve as predictors of alcohol consumption behaviors in young adults. Our study highlight the need for longitudinal research to evaluate the potential of taste- and smell-related brain connectivity patterns for early screening and targeted interventions, as well as their role in personalized treatment strategies for individuals at risk of AUD.
Q1

REM density predicts rapid antidepressant response to ketamine in individuals with treatment-resistant depression
Kheirkhah M., Duncan W.C., Yuan Q., Wang P.R., Jamalabadi H., Leistritz L., Walter M., Goldman D., Zarate C.A., Hejazi N.S.
Abstract
Abnormalities during rapid eye movement (REM) sleep contribute to the pathophysiology of major depressive disorder (MDD), but few studies have explored the relationship between REM sleep and treatment-resistant depression (TRD). In MDD, REM sleep abnormalities often manifest as alterations in total night REM Density (RD), RD in the first REM period (RD1), and REM Latency (RL). Among these, RD1 is notably considered a potential endophenotype of depression. This study compared REM sleep markers between 63 drug-free individuals with TRD (39 F/24 M) and 41 healthy volunteers (25 F/16 M). It also investigated the effects of ketamine, an N-methyl-D-aspartate (NMDA) receptor antagonist, on these REM sleep variables. Specifically, the study investigated whether RD1 could predict antidepressant response to ketamine. TRD participants showed higher RD1 and shorter RL at baseline compared to HVs, as assessed via non-parametric tests, but Total Night RD did not differ between the two groups. Ketamine treatment decreased RD1 in TRD participants but did not affect Total Night RD or RL. As assessed via the Support Vector Machine (SVM) algorithm, baseline RD1 level moderately predicted antidepressant response to ketamine versus non-response (area under the receiver operating characteristic (ROC) curve (AUC) = 0.73, with a median accuracy of 0.75), wherein TRD participants with higher baseline RD1 were more likely to respond to ketamine. These results underscore the utility of RD1 for identifying individuals most likely to benefit from ketamine treatment, enabling more targeted and effective therapeutic strategies. Clinical Trials Identifier: NCT00088699, NCT01204918.
Q1

Frontostriatal regulation of brain circuits contributes to flexible decision making
Duan Y., Ma Z., Tsai P., Lu H., Xiao X., Wang D., Siddiqi A., Stein E.A., Michaelides M., Yang Y.
Abstract
Deficits in behavioral or cognitive flexibility that are linked to altered activity in both cortical and subcortical brain regions, are often observed across multiple neuropsychiatric disorders. The medial prefrontal cortex (mPFC)-nucleus accumbens (NAc) pathway in rats plays a critical role in flexible control of behavior. However, the modulation of this pathway on activity and functional connectivity with the rest of the brain remains unclear. In this study, we first confirmed the role of the mPFC-NAc pathway in behavioral flexibility using a set-shifting task in rats and then evaluated the causal effects of mPFC-NAc activation induced by chemogenetic stimulation of the terminal axons of the NAc with DREADD expression on whole-brain activity and functional connectivity measured by functional MRI. mPFC-NAc activation improved performance on the set-shifting task by reducing perseverative errors. Additionally, stimulation of this pathway increased activity in a set of brain regions within the basal ganglia-thalamus-cortical loop network including NAc, thalamus, hypothalamus and various connected cortical regions, while also decreased functional connectivity strength of NAc-mPFC, NAc-secondary motor cortex (M2), and various cortical circuits. Moreover, performance on the set-shifting task was related to the functional connectivity strength of the above frontostriatal and cortical circuits. These findings provide insights into the link between specific frontostriatal circuits on decision making flexibility, which may inform potential future interventions for behavioral flexibility deficits.
Q1

Validation of L-type calcium channel blocker amlodipine as a novel ADHD treatment through cross-species analysis, drug-target Mendelian randomization, and clinical evidence from medical records
Þorsteinsson H., Baukmann H.A., Sveinsdóttir H.S., Halldórsdóttir D.Þ., Grzymala B., Hillman C., Rolfe-Tarrant J., Parker M.O., Cope J.L., Ravarani C.N., Schmidt M.F., Karlsson K.Æ.
Abstract
ADHD is a chronic neurodevelopmental disorder that significantly affects life outcomes, and current treatments often have adverse side effects, high abuse potential, and a 25% non-response rate, highlighting the need for new therapeutics. This study investigates amlodipine, an L-type calcium channel blocker, as a potential foundation for developing a novel ADHD treatment by integrating findings from animal models and human genetic data. Amlodipine reduced hyperactivity in SHR rats and decreased both hyperactivity and impulsivity in adgrl3.1−/− zebrafish. It also crosses the blood-brain barrier, reducing telencephalic activation. Crucially, Mendelian Randomization analysis linked ADHD to genetic variations in L-type calcium channel subunits (α1-C; CACNA1C, β1; CACNB1, α2δ3; CACNA2D3) targeted by amlodipine, while polygenic risk score analysis showed symptom mitigation in individuals with high ADHD genetic liability. With its well-tolerated profile and efficacy across species, supported by genetic evidence, amlodipine shows potential to be refined and developed into a novel treatment for ADHD.
Q1

Extended amygdala corticotropin-releasing hormone neurons regulate sexually dimorphic changes in pair bond formation following social defeat in prairie voles (Microtus ochrogaster)
Tickerhoof M.C., Nerio Morales L.K., Goff J., Vitale E.M., Smith A.S.
The neurobiological mechanisms underlying the connection between anxiety brought on by social stressors and the negative impact on relationship formation have remained elusive. In order to address this question, we used the social defeat model in the socially monogamous prairie vole to investigate the impact of this stress on pair bond formation. Social defeat experience inhibited partner preference formation in males but promoted preference in females. Furthermore, pair bonding increased corticotropin-releasing hormone (CRH) expression in the bed nucleus of the stria terminalis (BNST) in male prairie voles, while defeat experience increased BNST CRH expression in females. Chemogenetic excitation of BNST CRH neurons during a short cohabitation with a new partner promoted a partner preference in stress-naïve prairie voles. Interestingly, chemogenetic inhibition of BNST CRH neurons during cohabitation with a new partner blocked partner preference in stress-naïve males but promoted preference in defeated males. Inhibition of BNST CRH neurons also blocked partner preference in stress-naïve females but did not alter preference behavior in defeated females. This study revealed sexual dimorphism in not only the impact of social defeat on pair bond formation, but also in the role BNST CRHergic neurons play in regulating changes in pair bonding following social conflict.
Q1

Absence of TAAR1 function increases methamphetamine-induced excitability of dorsal raphe serotonin neurons and drives binge-level methamphetamine intake
Rios S.M., Mootz J.R., Phillips T.J., Ingram S.L.
Abstract
Methamphetamine (MA) is a potent psychostimulant capable of exerting both rewarding and aversive effects, the balance of which likely drives variation in voluntary MA intake. Understanding the genetic factors underlying sensitivity to these effects of MA is critical for developing effective treatments. The activity of dorsal raphe serotonin neurons is linked to reward processing. Here, we performed whole-cell patch-clamp electrophysiology in dorsal raphe serotonin neurons from mice with high or low MA intake corresponding with high or low MA reward sensitivity. The MA drinking (MADR) mice consist of the MA reward sensitive MA high drinking (MAHDR) and the MA reward insensitive MA low drinking (MALDR) lines. MA is a trace amine-associated receptor 1 (TAAR1) agonist, and MAHDR mice are homozygous for a mutation in the Taar1 gene, Taar1
m1J
, that encodes non-functional TAAR1, whereas MALDR mice possess at least one copy of the reference Taar1
+
allele that encodes functional TAAR1. Our previous research using CRISPR-Cas9-generated MAHDR-Taar1
+/+
knock-in mice in which Taar1
m1J
was replaced with Taar1
+
, and non-edited MAHDR-Taar1
m1J/m1J
controls demonstrated that lack of TAAR1 function is critical for heightened MA consumption and MA reward sensitivity. Here, electrophysiological recordings in the MADR lines demonstrate a MA-induced decrease in dorsal raphe serotonin neuron activity from MALDR, but not MAHDR mice. However, in the presence of serotonin autoreceptor antagonists, MA potentiates dorsal raphe serotonin neuron activity of MAHDR, but not MALDR mice. Importantly, potentiation in the presence of the antagonists is abolished in knock-in mice expressing functional TAAR1. The knock-in mice did not display binge-level MA intake, consistent with the loss of MA-reward sensitivity previously reported in mice with functional TAAR1. Finally, because MA is a substrate of the serotonin transporter, we evaluated whether the serotonin transporter is necessary for MA-induced potentiation of dorsal raphe serotonin neuron activity in mice with non-functional TAAR1. The serotonin transporter antagonist fluoxetine blocks MA-induced potentiation for both MAHDR and MAHDR-Taar1
m1J/m1J
mice. Thus, TAAR1 function directly impacts MA reward sensitivity and MA intake and serves as a critical regulator of MA-induced activity of dorsal raphe serotonin neurons through its interaction with the serotonin transporter.
Q1

A molecular mechanism mediating clozapine-enhanced sensorimotor gating
Mantas I., Flais I., Branzell N., Ionescu T.M., Kim E., Zhang X., Cash D., Hengerer B., Svenningsson P.
Abstract
The atypical antipsychotic clozapine targets multiple receptor systems beyond the dopaminergic pathway and influences prepulse inhibition (PPI), a critical translational measure of sensorimotor gating. Since PPI is modulated by atypical antipsychotics such as risperidone and clozapine, we hypothesized that p11—an adaptor protein associated with anxiety- and depressive-like behaviors and G-protein-coupled receptor function—might modulate these effects. In this study, we assessed the role of p11 in clozapine’s PPI-enhancing effect by testing wild-type and global p11 knockout (KO) mice in response to haloperidol, risperidone, and clozapine. We also performed structural and functional brain imaging. Contrary to our expectation that anxiety-like p11-KO mice would exhibit an augmented startle response and heightened sensitivity to clozapine, PPI tests showed that p11-KO mice were unresponsive to the PPI-enhancing effects of risperidone and clozapine. Imaging revealed distinct regional brain volume differences and reduced hippocampal connectivity in p11-KO mice, with significantly blunted clozapine-induced connectivity changes in the CA1 region. Our findings highlight a novel role for p11 in modulating clozapine’s effects on sensorimotor gating and hippocampal connectivity, offering new insight into its functional pathways.
Q1

Brain and cardiovascular responses to acute stress in remitted and recurrent late-life depression
Kraynak T.E., Karim H.T., Banihashemi L., Krafty R.T., Butters M.A., Ajilore O.A., Taylor W.D., Andreescu C.
In individuals with remitted late-life depression (LLD), stress exposure can increase the likelihood of a new, recurrent depressive episode. Variability in the effect of stress on recurrence risk may reflect underlying brain and physiological processes mediating the stress response. We examined how subjective, physiological, and brain responses to an experimental stressor differs in older adults with and without remitted depression, and how these stress responses relate to future relapse. Participants were recruited through 3 sites and included 76 older adults with remitted LLD and 36 age-matched healthy comparison (HC) adults. Participants completed an acute stressor task during functional brain imaging with behavioral and cardiovascular monitoring. Remitted LLD participants were followed longitudinally to evaluate depression recurrence. Compared to HC, the remitted LLD group exhibited reduced stressor-evoked systolic blood pressure and heart rate responses, as well as reduced stressor-evoked posterior insula activity. This blunted stress response phenotype appeared more specific to the stable remitter group than the relapsing LLD group. Survival analyses demonstrated that greater stressor-evoked bed nucleus of the stria terminalis (BNST) activity was associated with faster time to recurrence. These findings add to a growing literature reporting so-called “blunted” stressor-evoked cardiovascular and brain reactivity in remitted depression. Moreover, they link the stress response in visceral interoceptive brain circuits with relapse vulnerability. Future work involving longer follow-up periods may reveal additional stress-related brain and behavioral predictors of recurrence in remitted LLD.
Q1

N-acetylcysteine for youth cannabis use disorder: randomized controlled trial main findings
Gray K.M., Tomko R.L., Baker N.L., McClure E.A., McRae-Clark A.L., Squeglia L.M.
Abstract
Cannabis use disorder is particularly prevalent and impairing among young people, and evidence-based treatments are limited. Prior trials of N-acetylcysteine, added to contingency management as a platform behavioral intervention, yielded positive findings in youth but not in adults. This trial sought to rigorously evaluate whether N-acetylcysteine is efficacious in youth when not paired with a robust behavioral treatment platform. Treatment-seeking youth with cannabis use disorder (N = 192, ages 14–21) were randomized to receive a double-blind 12-week course of oral N-acetylcysteine 1200 mg or placebo twice daily; all received weekly medical management and brief behavioral counseling. The primary efficacy outcome was the proportion of negative urine cannabinoid tests during treatment, compared between groups. An array of self-report and urine testing measures were examined secondarily to assess cannabis use reduction and cessation outcomes. The N-acetylcysteine and placebo groups did not differ in proportion of negative urine cannabinoid tests (RR = 0.93, 95% CI = 0.53, 1.64; p = 0.80) or self-reported cannabis abstinence (RR = 1.02, 95% CI = 0.63, 1.65; p = 0.93) during treatment. The mean percentage of cannabis use days and grams of cannabis used per using day decreased over time during treatment but did not differ between groups. More N-acetylcysteine than placebo treated participants reported gastrointestinal adverse events (63/98 versus 37/94, χ2
1 = 11.9 p < 0.001); adverse events were otherwise similar between groups. Findings indicate that N-acetylcysteine is not efficacious for youth cannabis use disorder when not paired with contingency management, highlighting the potentially crucial role of a robust behavioral treatment platform in facilitating prior positive efficacy findings with N-acetylcysteine.
Trial Registration: Clinicaltrials.gov identifier NCT03055377
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Frontiers in Psychiatry
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|
BMJ
21 citations, 0.6%
|
|
Bentham Science Publishers Ltd.
17 citations, 0.49%
|
|
European Society of Traumatic Stress Studies (ESTSS)
17 citations, 0.49%
|
|
SLACK
16 citations, 0.46%
|
|
Institute of Electrical and Electronics Engineers (IEEE)
12 citations, 0.35%
|
|
American Medical Association (AMA)
11 citations, 0.32%
|
|
S. Karger AG
11 citations, 0.32%
|
|
F1000 Research
11 citations, 0.32%
|
|
Association for Computing Machinery (ACM)
9 citations, 0.26%
|
|
IntechOpen
9 citations, 0.26%
|
|
Emerald
8 citations, 0.23%
|
|
Hindawi Limited
8 citations, 0.23%
|
|
Guilford Publications
8 citations, 0.23%
|
|
Georg Thieme Verlag KG
7 citations, 0.2%
|
|
Annual Reviews
7 citations, 0.2%
|
|
Baishideng Publishing Group
7 citations, 0.2%
|
|
Walter de Gruyter
6 citations, 0.17%
|
|
Royal College of Psychiatrists
6 citations, 0.17%
|
|
Jaypee Brothers Medical Publishing
6 citations, 0.17%
|
|
Korean Neuropsychiatric Association
5 citations, 0.14%
|
|
Scientific Research Publishing
5 citations, 0.14%
|
|
University of Toronto Press Inc. (UTPress)
5 citations, 0.14%
|
|
Proceedings of the National Academy of Sciences (PNAS)
4 citations, 0.12%
|
|
Society for Neuroscience
4 citations, 0.12%
|
|
Medknow
4 citations, 0.12%
|
|
AOSIS
4 citations, 0.12%
|
|
Hogrefe Publishing Group
4 citations, 0.12%
|
|
Research Square Platform LLC
4 citations, 0.12%
|
|
IOS Press
3 citations, 0.09%
|
|
The Royal Society
3 citations, 0.09%
|
|
PeerJ
3 citations, 0.09%
|
|
Association of Military Surgeons of the US
3 citations, 0.09%
|
|
Mark Allen Group
3 citations, 0.09%
|
|
American Psychological Association (APA)
3 citations, 0.09%
|
|
Akademiai Kiado
3 citations, 0.09%
|
|
Medical Informational Agency Publishers
3 citations, 0.09%
|
|
Media Sphere Publishing House
3 citations, 0.09%
|
|
Tech Science Press
3 citations, 0.09%
|
|
American Association for the Advancement of Science (AAAS)
2 citations, 0.06%
|
|
Les Laboratoires Servier
2 citations, 0.06%
|
|
American Society for Pharmacology and Experimental Therapeutics
2 citations, 0.06%
|
|
American Academy of Pediatrics
2 citations, 0.06%
|
|
Massachusetts Medical Society
2 citations, 0.06%
|
|
Associacao Brasileira de Psiquiatria
2 citations, 0.06%
|
|
2 citations, 0.06%
|
|
SciELO
2 citations, 0.06%
|
|
American Chemical Society (ACS)
1 citation, 0.03%
|
|
John Benjamins Publishing Company
1 citation, 0.03%
|
|
Pleiades Publishing
1 citation, 0.03%
|
|
Royal Society of Chemistry (RSC)
1 citation, 0.03%
|
|
American Physiological Society
1 citation, 0.03%
|
|
AIP Publishing
1 citation, 0.03%
|
|
Springer Publishing Company
1 citation, 0.03%
|
|
1 citation, 0.03%
|
|
American Scientific Publishers
1 citation, 0.03%
|
|
Health Affairs (Project Hope)
1 citation, 0.03%
|
|
Eco-Vector LLC
1 citation, 0.03%
|
|
AME Publishing Company
1 citation, 0.03%
|
|
American College of Physicians
1 citation, 0.03%
|
|
IOP Publishing
1 citation, 0.03%
|
|
American Association for Clinical Chemistry
1 citation, 0.03%
|
|
Technosdar Ltd
1 citation, 0.03%
|
|
Revue Sante Mentale au Quebec
1 citation, 0.03%
|
|
U.S. Agency for International Development
1 citation, 0.03%
|
|
Indian Association for Child and Adolescent Mental Health
1 citation, 0.03%
|
|
American Society of Health-System Pharmacists
1 citation, 0.03%
|
|
1 citation, 0.03%
|
|
Biola University
1 citation, 0.03%
|
|
National Coordinating Centre for Health Technology Assessment
1 citation, 0.03%
|
|
Korean College of Neuropsychopharmacology
1 citation, 0.03%
|
|
American Music Therapy Association
1 citation, 0.03%
|
|
Korean Academy of Medical Sciences
1 citation, 0.03%
|
|
China Science Publishing & Media
1 citation, 0.03%
|
|
Canadian Science Publishing
1 citation, 0.03%
|
|
Academy of Managed Care Pharmacy
1 citation, 0.03%
|
|
Social Science Electronic Publishing
1 citation, 0.03%
|
|
IMA Press, LLC
1 citation, 0.03%
|
|
The Genetics Society of Japan
1 citation, 0.03%
|
|
MedCrave Group Kft.
1 citation, 0.03%
|
|
V.M. Bekhterev National Research Medical Center for Psychiatry and Neurology
1 citation, 0.03%
|
|
College of Psychiatric and Neurologic Pharmacists
1 citation, 0.03%
|
|
Paediatrician Publishers LLC
1 citation, 0.03%
|
|
PANORAMA Publishing House
1 citation, 0.03%
|
|
RCNi
1 citation, 0.03%
|
|
Show all (70 more) | |
100
200
300
400
500
600
700
800
|
Publishing organizations
5
10
15
20
25
30
|
|
Harvard University
28 publications, 8.26%
|
|
Boston University
19 publications, 5.6%
|
|
Icahn School of Medicine at Mount Sinai
16 publications, 4.72%
|
|
Stanford University
15 publications, 4.42%
|
|
Virginia Commonwealth University
15 publications, 4.42%
|
|
Yale University
12 publications, 3.54%
|
|
University of California, San Diego
11 publications, 3.24%
|
|
University of Minnesota
11 publications, 3.24%
|
|
Brown University
11 publications, 3.24%
|
|
Northwestern University
10 publications, 2.95%
|
|
University of Toronto
10 publications, 2.95%
|
|
University of California, San Francisco
9 publications, 2.65%
|
|
University of Cincinnati
9 publications, 2.65%
|
|
University of Colorado Anschutz Medical Campus
8 publications, 2.36%
|
|
Columbia University
7 publications, 2.06%
|
|
University of Texas at Austin
7 publications, 2.06%
|
|
University of Lisbon
6 publications, 1.77%
|
|
Cornell University
6 publications, 1.77%
|
|
Johns Hopkins University
6 publications, 1.77%
|
|
Massachusetts General Hospital
6 publications, 1.77%
|
|
Dartmouth College
6 publications, 1.77%
|
|
University of Miami
6 publications, 1.77%
|
|
University of Melbourne
5 publications, 1.47%
|
|
University of Texas Southwestern Medical Center
5 publications, 1.47%
|
|
Centre for Addiction and Mental Health
5 publications, 1.47%
|
|
Beth Israel Deaconess Medical Center
5 publications, 1.47%
|
|
Pennsylvania State University
4 publications, 1.18%
|
|
University of Illinois at Chicago
4 publications, 1.18%
|
|
New York University
4 publications, 1.18%
|
|
University of Washington
4 publications, 1.18%
|
|
University of California, Los Angeles
4 publications, 1.18%
|
|
University of British Columbia
4 publications, 1.18%
|
|
University of Pennsylvania
4 publications, 1.18%
|
|
University of North Carolina at Chapel Hill
4 publications, 1.18%
|
|
Cincinnati Children's Hospital Medical Center
4 publications, 1.18%
|
|
University of Connecticut
4 publications, 1.18%
|
|
Washington University in St. Louis
3 publications, 0.88%
|
|
Duke University
3 publications, 0.88%
|
|
Brigham and Women's Hospital
3 publications, 0.88%
|
|
University of California, Irvine
3 publications, 0.88%
|
|
Emory University
3 publications, 0.88%
|
|
Temple University
3 publications, 0.88%
|
|
Mayo Clinic
3 publications, 0.88%
|
|
Western University
3 publications, 0.88%
|
|
University of Kentucky
3 publications, 0.88%
|
|
Centro de Investigación Biomédica en Red de Salud Mental
3 publications, 0.88%
|
|
University of Colorado Colorado Springs
3 publications, 0.88%
|
|
University of Texas at Arlington
3 publications, 0.88%
|
|
University of Connecticut Health
3 publications, 0.88%
|
|
Radboud University Nijmegen
2 publications, 0.59%
|
|
Università Cattolica del Sacro Cuore
2 publications, 0.59%
|
|
University of Turin
2 publications, 0.59%
|
|
Balamand University
2 publications, 0.59%
|
|
University of Copenhagen
2 publications, 0.59%
|
|
King's College London
2 publications, 0.59%
|
|
Florida State University
2 publications, 0.59%
|
|
University of Sydney
2 publications, 0.59%
|
|
Georgia Institute of technology
2 publications, 0.59%
|
|
University of Queensland
2 publications, 0.59%
|
|
Swinburne University of Technology
2 publications, 0.59%
|
|
University of Cape Town
2 publications, 0.59%
|
|
Virginia Tech
2 publications, 0.59%
|
|
Case Western Reserve University
2 publications, 0.59%
|
|
Ohio State University
2 publications, 0.59%
|
|
Tufts University
2 publications, 0.59%
|
|
Priscilla Chan and Mark Zuckerberg San Francisco General Hospital and Trauma Center
2 publications, 0.59%
|
|
University of California, Davis
2 publications, 0.59%
|
|
Federal University of Santa Catarina
2 publications, 0.59%
|
|
Vrije Universiteit Medical Center
2 publications, 0.59%
|
|
University of Michigan
2 publications, 0.59%
|
|
McMaster University
2 publications, 0.59%
|
|
Dartmouth–Hitchcock Medical Center
2 publications, 0.59%
|
|
Memorial Sloan Kettering Cancer Center
2 publications, 0.59%
|
|
University of Maryland, Baltimore County
2 publications, 0.59%
|
|
Uniformed Services University of the Health Sciences
2 publications, 0.59%
|
|
Fordham University
2 publications, 0.59%
|
|
Toronto Metropolitan University
2 publications, 0.59%
|
|
University of Calgary
2 publications, 0.59%
|
|
University of Ottawa
2 publications, 0.59%
|
|
Wright State University
2 publications, 0.59%
|
|
Miami University
2 publications, 0.59%
|
|
University of Texas at Tyler
2 publications, 0.59%
|
|
NewYork-Presbyterian Hospital
2 publications, 0.59%
|
|
National Institute on Drug Abuse
2 publications, 0.59%
|
|
University of Nebraska Medical Center
2 publications, 0.59%
|
|
First Pavlov State Medical University of St. Petersburg
1 publication, 0.29%
|
|
Bekhterev National Medical Research Center for Psychiatry and Neurology
1 publication, 0.29%
|
|
Ege University
1 publication, 0.29%
|
|
All India Institute of Medical Sciences, Bhopal
1 publication, 0.29%
|
|
Dokuz Eylül University
1 publication, 0.29%
|
|
Aga Khan University Hospital
1 publication, 0.29%
|
|
Jinnah Sindh Medical University
1 publication, 0.29%
|
|
Vietnam National University, Hanoi
1 publication, 0.29%
|
|
RMIT Vietnam
1 publication, 0.29%
|
|
National Institute of Mental Health and Neurosciences
1 publication, 0.29%
|
|
Post graduate Institute of Medical Education and Research
1 publication, 0.29%
|
|
Peking University
1 publication, 0.29%
|
|
Shanghai Jiao Tong University
1 publication, 0.29%
|
|
Tel Aviv University
1 publication, 0.29%
|
|
Weizmann Institute of Science
1 publication, 0.29%
|
|
Show all (70 more) | |
5
10
15
20
25
30
|
Publishing organizations in 5 years
2
4
6
8
10
12
14
16
|
|
Harvard University
15 publications, 10.27%
|
|
Boston University
10 publications, 6.85%
|
|
Stanford University
8 publications, 5.48%
|
|
University of Minnesota
8 publications, 5.48%
|
|
University of California, San Francisco
7 publications, 4.79%
|
|
Brown University
7 publications, 4.79%
|
|
University of Colorado Anschutz Medical Campus
7 publications, 4.79%
|
|
University of Lisbon
6 publications, 4.11%
|
|
University of California, San Diego
6 publications, 4.11%
|
|
Yale University
4 publications, 2.74%
|
|
Northwestern University
4 publications, 2.74%
|
|
University of Texas at Austin
4 publications, 2.74%
|
|
University of Texas Southwestern Medical Center
4 publications, 2.74%
|
|
University of Toronto
4 publications, 2.74%
|
|
University of California, Los Angeles
3 publications, 2.05%
|
|
Icahn School of Medicine at Mount Sinai
3 publications, 2.05%
|
|
Dartmouth College
3 publications, 2.05%
|
|
University of Colorado Colorado Springs
3 publications, 2.05%
|
|
University of Texas at Arlington
3 publications, 2.05%
|
|
University of Connecticut
3 publications, 2.05%
|
|
Beth Israel Deaconess Medical Center
3 publications, 2.05%
|
|
Università Cattolica del Sacro Cuore
2 publications, 1.37%
|
|
University of Turin
2 publications, 1.37%
|
|
Balamand University
2 publications, 1.37%
|
|
University of Queensland
2 publications, 1.37%
|
|
Washington University in St. Louis
2 publications, 1.37%
|
|
Duke University
2 publications, 1.37%
|
|
Massachusetts General Hospital
2 publications, 1.37%
|
|
Tufts University
2 publications, 1.37%
|
|
University of California, Davis
2 publications, 1.37%
|
|
University of California, Irvine
2 publications, 1.37%
|
|
Federal University of Santa Catarina
2 publications, 1.37%
|
|
University of British Columbia
2 publications, 1.37%
|
|
Dartmouth–Hitchcock Medical Center
2 publications, 1.37%
|
|
Virginia Commonwealth University
2 publications, 1.37%
|
|
Emory University
2 publications, 1.37%
|
|
Temple University
2 publications, 1.37%
|
|
Mayo Clinic
2 publications, 1.37%
|
|
University of Pennsylvania
2 publications, 1.37%
|
|
Western University
2 publications, 1.37%
|
|
University of Texas at Tyler
2 publications, 1.37%
|
|
University of Nebraska Medical Center
2 publications, 1.37%
|
|
University of Connecticut Health
2 publications, 1.37%
|
|
All India Institute of Medical Sciences, Bhopal
1 publication, 0.68%
|
|
Aga Khan University Hospital
1 publication, 0.68%
|
|
Jinnah Sindh Medical University
1 publication, 0.68%
|
|
Vietnam National University, Hanoi
1 publication, 0.68%
|
|
RMIT Vietnam
1 publication, 0.68%
|
|
National Institute of Mental Health and Neurosciences
1 publication, 0.68%
|
|
Hebrew University of Jerusalem
1 publication, 0.68%
|
|
Shaare Zedek Medical Center
1 publication, 0.68%
|
|
Uppsala University
1 publication, 0.68%
|
|
Radboud University Nijmegen
1 publication, 0.68%
|
|
University of Bologna
1 publication, 0.68%
|
|
American University of Beirut
1 publication, 0.68%
|
|
Beirut Arab University
1 publication, 0.68%
|
|
University of Copenhagen
1 publication, 0.68%
|
|
Florida State University
1 publication, 0.68%
|
|
University of Southern California
1 publication, 0.68%
|
|
Cornell University
1 publication, 0.68%
|
|
Johns Hopkins University
1 publication, 0.68%
|
|
University of Sydney
1 publication, 0.68%
|
|
Georgia Institute of technology
1 publication, 0.68%
|
|
Pennsylvania State University
1 publication, 0.68%
|
|
Iowa State University
1 publication, 0.68%
|
|
University of Melbourne
1 publication, 0.68%
|
|
La Trobe University
1 publication, 0.68%
|
|
Columbia University
1 publication, 0.68%
|
|
Georgetown University
1 publication, 0.68%
|
|
University of Illinois at Chicago
1 publication, 0.68%
|
|
Rutgers, The State University of New Jersey
1 publication, 0.68%
|
|
George Mason University
1 publication, 0.68%
|
|
Oregon State University
1 publication, 0.68%
|
|
Oregon Health & Science University
1 publication, 0.68%
|
|
Case Western Reserve University
1 publication, 0.68%
|
|
Towson University
1 publication, 0.68%
|
|
New York University
1 publication, 0.68%
|
|
Brigham and Women's Hospital
1 publication, 0.68%
|
|
Priscilla Chan and Mark Zuckerberg San Francisco General Hospital and Trauma Center
1 publication, 0.68%
|
|
University of Texas at Dallas
1 publication, 0.68%
|
|
Albert Einstein College of Medicine
1 publication, 0.68%
|
|
Agency for Science, Technology and Research
1 publication, 0.68%
|
|
University of Michigan
1 publication, 0.68%
|
|
Thompson Rivers University
1 publication, 0.68%
|
|
McMaster University
1 publication, 0.68%
|
|
Queen's University at Kingston
1 publication, 0.68%
|
|
Fraunhofer USA Center Mid-Atlantic CMA
1 publication, 0.68%
|
|
Federal University of Rio Grande do Sul
1 publication, 0.68%
|
|
University of Maryland, Baltimore County
1 publication, 0.68%
|
|
Uniformed Services University of the Health Sciences
1 publication, 0.68%
|
|
Baylor University
1 publication, 0.68%
|
|
Toronto Metropolitan University
1 publication, 0.68%
|
|
Wilfrid Laurier University
1 publication, 0.68%
|
|
University of Alberta
1 publication, 0.68%
|
|
Florida International University
1 publication, 0.68%
|
|
University of Ottawa
1 publication, 0.68%
|
|
Wright State University
1 publication, 0.68%
|
|
University of Florida
1 publication, 0.68%
|
|
University of North Carolina at Greensboro
1 publication, 0.68%
|
|
University of North Carolina at Chapel Hill
1 publication, 0.68%
|
|
Show all (70 more) | |
2
4
6
8
10
12
14
16
|
Publishing countries
50
100
150
200
250
300
|
|
USA
|
USA, 271, 79.94%
USA
271 publications, 79.94%
|
Canada
|
Canada, 24, 7.08%
Canada
24 publications, 7.08%
|
Australia
|
Australia, 12, 3.54%
Australia
12 publications, 3.54%
|
Portugal
|
Portugal, 6, 1.77%
Portugal
6 publications, 1.77%
|
Netherlands
|
Netherlands, 6, 1.77%
Netherlands
6 publications, 1.77%
|
Brazil
|
Brazil, 4, 1.18%
Brazil
4 publications, 1.18%
|
Italy
|
Italy, 4, 1.18%
Italy
4 publications, 1.18%
|
Germany
|
Germany, 3, 0.88%
Germany
3 publications, 0.88%
|
United Kingdom
|
United Kingdom, 3, 0.88%
United Kingdom
3 publications, 0.88%
|
Israel
|
Israel, 3, 0.88%
Israel
3 publications, 0.88%
|
India
|
India, 3, 0.88%
India
3 publications, 0.88%
|
Spain
|
Spain, 3, 0.88%
Spain
3 publications, 0.88%
|
Lebanon
|
Lebanon, 3, 0.88%
Lebanon
3 publications, 0.88%
|
France
|
France, 2, 0.59%
France
2 publications, 0.59%
|
China
|
China, 2, 0.59%
China
2 publications, 0.59%
|
Denmark
|
Denmark, 2, 0.59%
Denmark
2 publications, 0.59%
|
New Zealand
|
New Zealand, 2, 0.59%
New Zealand
2 publications, 0.59%
|
Norway
|
Norway, 2, 0.59%
Norway
2 publications, 0.59%
|
Singapore
|
Singapore, 2, 0.59%
Singapore
2 publications, 0.59%
|
Turkey
|
Turkey, 2, 0.59%
Turkey
2 publications, 0.59%
|
South Africa
|
South Africa, 2, 0.59%
South Africa
2 publications, 0.59%
|
Japan
|
Japan, 2, 0.59%
Japan
2 publications, 0.59%
|
Russia
|
Russia, 1, 0.29%
Russia
1 publication, 0.29%
|
Austria
|
Austria, 1, 0.29%
Austria
1 publication, 0.29%
|
Bulgaria
|
Bulgaria, 1, 0.29%
Bulgaria
1 publication, 0.29%
|
Vietnam
|
Vietnam, 1, 0.29%
Vietnam
1 publication, 0.29%
|
Ireland
|
Ireland, 1, 0.29%
Ireland
1 publication, 0.29%
|
Pakistan
|
Pakistan, 1, 0.29%
Pakistan
1 publication, 0.29%
|
Switzerland
|
Switzerland, 1, 0.29%
Switzerland
1 publication, 0.29%
|
Sweden
|
Sweden, 1, 0.29%
Sweden
1 publication, 0.29%
|
50
100
150
200
250
300
|
Publishing countries in 5 years
20
40
60
80
100
120
|
|
USA
|
USA, 109, 74.66%
USA
109 publications, 74.66%
|
Canada
|
Canada, 10, 6.85%
Canada
10 publications, 6.85%
|
Portugal
|
Portugal, 6, 4.11%
Portugal
6 publications, 4.11%
|
Australia
|
Australia, 6, 4.11%
Australia
6 publications, 4.11%
|
Italy
|
Italy, 3, 2.05%
Italy
3 publications, 2.05%
|
Lebanon
|
Lebanon, 3, 2.05%
Lebanon
3 publications, 2.05%
|
Brazil
|
Brazil, 2, 1.37%
Brazil
2 publications, 1.37%
|
Vietnam
|
Vietnam, 1, 0.68%
Vietnam
1 publication, 0.68%
|
Denmark
|
Denmark, 1, 0.68%
Denmark
1 publication, 0.68%
|
Israel
|
Israel, 1, 0.68%
Israel
1 publication, 0.68%
|
India
|
India, 1, 0.68%
India
1 publication, 0.68%
|
Netherlands
|
Netherlands, 1, 0.68%
Netherlands
1 publication, 0.68%
|
New Zealand
|
New Zealand, 1, 0.68%
New Zealand
1 publication, 0.68%
|
Norway
|
Norway, 1, 0.68%
Norway
1 publication, 0.68%
|
Pakistan
|
Pakistan, 1, 0.68%
Pakistan
1 publication, 0.68%
|
Singapore
|
Singapore, 1, 0.68%
Singapore
1 publication, 0.68%
|
Sweden
|
Sweden, 1, 0.68%
Sweden
1 publication, 0.68%
|
20
40
60
80
100
120
|