Dialogue-Canadian Philosophical Review
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SCImago
Q2
Impact factor
0.2
SJR
0.157
CiteScore
0.5
Categories
Philosophy
Areas
Arts and Humanities
Years of issue
1962-2025
journal names
Dialogue-Canadian Philosophical Review
Dialogue
DIALOGUE-CAN PHILOS
Top-3 citing journals

Dialogue-Canadian Philosophical Review
(356 citations)

Synthese
(98 citations)

SSRN Electronic Journal
(80 citations)
Top-3 organizations

University of Ottawa
(278 publications)

Queen's University at Kingston
(149 publications)

Université du Québec à Montréal
(134 publications)

York University
(8 publications)

University of Toronto
(6 publications)

McGill University
(5 publications)
Most cited in 5 years
Found
Publications found: 8233
Q1

Central Executive Network drives delta-9-tetrahydrocannabinol (THC)-induced nonlinear changes in large-scale functional connectivity in adolescent nonhuman primates
Byun A.J., Deshpande H.U., Stover J., Kangas B.D., Kohut S.J.
Q1
Neuropsychopharmacology
,
2025
,
citations by CoLab: 0

Q1

A new module in the drug development process: preclinical multi-center randomized controlled trial of R-ketamine on alcohol relapse
Meinhardt M.W., Skorodumov I., Jeanblanc J., Benvenuti F., Hilal F.F., Domi E., André C., Bodeau S., Jeanblanc V., Domanegg K., Ciccocioppo R., Naassila M., Spanagel R.
Abstract
The drug development process in psychiatry faces significant challenges due to low reproducibility rates in animal testing, which often leads to translation failures. To address this issue, we introduce a new approach in psychiatric drug development: a preclinical randomized controlled trial (preRCT). To demonstrate its potential utility, we conducted a multi-center preRCT using the alcohol deprivation effect (ADE) model to assess the impact of ketamine and R-ketamine on alcohol relapse across three European research centers. Ketamine (20 mg/kg) significantly reduced relapse, while R-ketamine showed efficacy only in females. A higher dose of R-ketamine (40 mg/kg) was also effective in males. These sex-dependent effects were linked to plasma R-ketamine levels, which were two-fold higher in female compared to male rats. Notably, R-ketamine demonstrated a lasting reduction in alcohol consumption without adverse effects. In conclusion, our preRCT demonstrates R-ketamine’s effectiveness in reducing alcohol relapse and supports translation to a clinical RCT that accounts for sex-dependent effects.
Q1

The crosstalk between CREB and PER2 mediates the transition between mania- and depression-like behavior
Wang X., Ji Y., Li S., Serchov T.
Abstract
Bipolar disorder (BD) is a severe psychiatric disorder characterized by alternating manic and depressive episodes. The molecular mechanisms underlying the transition between mania and depression remain unclear. Utilizing a mania animal model induced by ouabain, we observed reduced phosphorylated level of cyclic AMP-responsive element-binding protein (pCREB) and Period (PER)2 expression in the cornu ammonis (CA1) region of the hippocampus, which were restored by lithium treatment. shRNA knockdown of CREB or Per2 in CA1 region induced mania-like behavior, while overexpression of both factors resulted in depression-like behavior. Furthermore, our protein analyses revealed that the upregulation or downregulation of CREB or Per2 influenced each other’s expression. Co-immunoprecipitation results demonstrated that CREB interacts with PER2. Taken together, our data suggest for potential inter-regulatory crosstalk between CREB–PER2 in hippocampal CA1 region, which mediates the transition between mania- and depression-like behaviors.
Q1

Pharmacological reduction of reverse-translated hippocampal hyperactivity in mouse: relevance for psychosis
Dybowski F.P., Scott D.S., Tamminga C.A.
Q1
Neuropsychopharmacology
,
2025
,
citations by CoLab: 0

Q1

Longitudinal associations between white matter integrity, early life adversities, and treatment response following cognitive-behavioral therapy in depression
Flinkenflügel K., Borgers T., Klug M., Mummendey M.M., Leehr E.J., Meinert S., Gruber M., Repple J., Kircher T., Opel N., Bauer J., Zwiky E., König P., Küttner A., Schöniger K., et. al.
Abstract
Cognitive-behavioral therapy (CBT) is a primary treatment for depression. Although previous research has underscored the significant roles of white matter (WM) alterations and maladaptive parenting in depression risk, their associations with CBT response remain largely unknown. This longitudinal study investigated the interplay of WM integrity changes over time, treatment response, and parenting style in patients with depression. Diffusion-tensor-imaging and clinical data were assessed in n = 65 (55% female) patients with depression before and after 20 CBT sessions and n = 65 (68% female) healthy controls (HC) in a naturalistic design. Linear-mixed-effect models compared changes in fractional anisotropy (FA) between groups and tested associations between FA changes and symptom changes. It was investigated whether parenting style predicts depressive symptoms at follow-up and whether FA changes mediate this association. Patients showed differential FA changes over time in the corpus callosum and corona radiata compared to HC (p
tfce-FWE = 0.008). Increases in FA in the corpus callosum, corona radiata and superior longitudinal fasciculus were linked to symptom improvement after CBT in patients (p
tfce-FWE = 0.023). High parental care (p
FDR = 0.010) and low maternal overprotection (p
FDR = 0.001) predicted fewer depressive symptoms at follow-up. The association between maternal overprotection and depressive symptoms at follow-up was mediated by FA changes (p
FDR = 0.044). Robustness checks—controlling for outliers, non-linear age effects, clinical characteristics, and patient subgroups—supported these results. Overall, patients with depression show changes in WM integrity following CBT, which are linked to treatment response. The results highlight the significance of early life adversities and related microstructural changes in the effectiveness of CBT for treating depression.
Q1

Protein interacting with C-kinase 1 (PICK1) regulates synaptic function and reversal learning in a mouse model for schizophrenia
Samsom J.N., Xu M., Ávila A., Daskalakis A.A., Dai J.H., Gao X., Georgiou J., Collingridge G.L., Liu F., Wong A.H.
Q1
Neuropsychopharmacology
,
2025
,
citations by CoLab: 0

Q1

Prediction of alcohol intake patterns with olfactory and gustatory brain connectivity networks
Agarwal K., Chaudhary S., Tomasi D., Volkow N.D., Joseph P.V.
Abstract
Craving in alcohol drinkers is often triggered by chemosensory cues, such as taste and smell, which are linked to brain network connectivity. This study aimed to investigate whether these brain connectivity patterns could predict alcohol intake in young adults. Resting-state fMRI data were obtained from the Human Connectome Project (HCP) Young Adult cohort, comprising 1003 participants. Functional connectomes generated from 100 independent components were analyzed, identifying significant connections correlated with taste and odor scores after applying a false discovery rate (FDR) correction using the Benjamini-Hochberg (BH) method. These significant connections were then utilized as predictors in general linear models for various alcohol intake metrics. The models were validated in an independent sample to assess their accuracy. The training sample (n = 702) and the validation sample (n = 117) showed no significant demographic differences. Out of 742 possible connections, 41 related to odor and 25 related to taste passed the significance threshold (P < 0.05) after FDR-BH correction. Notable predictors included visual-visual connectivity (node32-node13: β = 0.028, P = 0.02) for wine consumption and connectivity between the ventral attention network (VAN) and the frontal parietal/caudate nucleus (FP/CN) (node27-node9: β = −0.31, P = 0.04) for total alcohol intake in the past-week and maximum number of drinks per day in the past-year. The predictive models demonstrated strong accuracy, with root mean square error (RMSE) values of 5.15 for odor-related models and 5.14 for taste-related models. The F1 scores were 0.74 for the odor model and 0.71 for the taste model, indicating reliable performance. These findings suggest that specific patterns of brain connectivity associated with taste and olfactory perception may serve as predictors of alcohol consumption behaviors in young adults. Our study highlight the need for longitudinal research to evaluate the potential of taste- and smell-related brain connectivity patterns for early screening and targeted interventions, as well as their role in personalized treatment strategies for individuals at risk of AUD.
Q1

REM density predicts rapid antidepressant response to ketamine in individuals with treatment-resistant depression
Kheirkhah M., Duncan W.C., Yuan Q., Wang P.R., Jamalabadi H., Leistritz L., Walter M., Goldman D., Zarate C.A., Hejazi N.S.
Abstract
Abnormalities during rapid eye movement (REM) sleep contribute to the pathophysiology of major depressive disorder (MDD), but few studies have explored the relationship between REM sleep and treatment-resistant depression (TRD). In MDD, REM sleep abnormalities often manifest as alterations in total night REM Density (RD), RD in the first REM period (RD1), and REM Latency (RL). Among these, RD1 is notably considered a potential endophenotype of depression. This study compared REM sleep markers between 63 drug-free individuals with TRD (39 F/24 M) and 41 healthy volunteers (25 F/16 M). It also investigated the effects of ketamine, an N-methyl-D-aspartate (NMDA) receptor antagonist, on these REM sleep variables. Specifically, the study investigated whether RD1 could predict antidepressant response to ketamine. TRD participants showed higher RD1 and shorter RL at baseline compared to HVs, as assessed via non-parametric tests, but Total Night RD did not differ between the two groups. Ketamine treatment decreased RD1 in TRD participants but did not affect Total Night RD or RL. As assessed via the Support Vector Machine (SVM) algorithm, baseline RD1 level moderately predicted antidepressant response to ketamine versus non-response (area under the receiver operating characteristic (ROC) curve (AUC) = 0.73, with a median accuracy of 0.75), wherein TRD participants with higher baseline RD1 were more likely to respond to ketamine. These results underscore the utility of RD1 for identifying individuals most likely to benefit from ketamine treatment, enabling more targeted and effective therapeutic strategies. Clinical Trials Identifier: NCT00088699, NCT01204918.
Q1

Frontostriatal regulation of brain circuits contributes to flexible decision making
Duan Y., Ma Z., Tsai P., Lu H., Xiao X., Wang D., Siddiqi A., Stein E.A., Michaelides M., Yang Y.
Abstract
Deficits in behavioral or cognitive flexibility that are linked to altered activity in both cortical and subcortical brain regions, are often observed across multiple neuropsychiatric disorders. The medial prefrontal cortex (mPFC)-nucleus accumbens (NAc) pathway in rats plays a critical role in flexible control of behavior. However, the modulation of this pathway on activity and functional connectivity with the rest of the brain remains unclear. In this study, we first confirmed the role of the mPFC-NAc pathway in behavioral flexibility using a set-shifting task in rats and then evaluated the causal effects of mPFC-NAc activation induced by chemogenetic stimulation of the terminal axons of the NAc with DREADD expression on whole-brain activity and functional connectivity measured by functional MRI. mPFC-NAc activation improved performance on the set-shifting task by reducing perseverative errors. Additionally, stimulation of this pathway increased activity in a set of brain regions within the basal ganglia-thalamus-cortical loop network including NAc, thalamus, hypothalamus and various connected cortical regions, while also decreased functional connectivity strength of NAc-mPFC, NAc-secondary motor cortex (M2), and various cortical circuits. Moreover, performance on the set-shifting task was related to the functional connectivity strength of the above frontostriatal and cortical circuits. These findings provide insights into the link between specific frontostriatal circuits on decision making flexibility, which may inform potential future interventions for behavioral flexibility deficits.
Q1

Validation of L-type calcium channel blocker amlodipine as a novel ADHD treatment through cross-species analysis, drug-target Mendelian randomization, and clinical evidence from medical records
Þorsteinsson H., Baukmann H.A., Sveinsdóttir H.S., Halldórsdóttir D.Þ., Grzymala B., Hillman C., Rolfe-Tarrant J., Parker M.O., Cope J.L., Ravarani C.N., Schmidt M.F., Karlsson K.Æ.
Abstract
ADHD is a chronic neurodevelopmental disorder that significantly affects life outcomes, and current treatments often have adverse side effects, high abuse potential, and a 25% non-response rate, highlighting the need for new therapeutics. This study investigates amlodipine, an L-type calcium channel blocker, as a potential foundation for developing a novel ADHD treatment by integrating findings from animal models and human genetic data. Amlodipine reduced hyperactivity in SHR rats and decreased both hyperactivity and impulsivity in adgrl3.1−/− zebrafish. It also crosses the blood-brain barrier, reducing telencephalic activation. Crucially, Mendelian Randomization analysis linked ADHD to genetic variations in L-type calcium channel subunits (α1-C; CACNA1C, β1; CACNB1, α2δ3; CACNA2D3) targeted by amlodipine, while polygenic risk score analysis showed symptom mitigation in individuals with high ADHD genetic liability. With its well-tolerated profile and efficacy across species, supported by genetic evidence, amlodipine shows potential to be refined and developed into a novel treatment for ADHD.
Q1

Extended amygdala corticotropin-releasing hormone neurons regulate sexually dimorphic changes in pair bond formation following social defeat in prairie voles (Microtus ochrogaster)
Tickerhoof M.C., Nerio Morales L.K., Goff J., Vitale E.M., Smith A.S.
The neurobiological mechanisms underlying the connection between anxiety brought on by social stressors and the negative impact on relationship formation have remained elusive. In order to address this question, we used the social defeat model in the socially monogamous prairie vole to investigate the impact of this stress on pair bond formation. Social defeat experience inhibited partner preference formation in males but promoted preference in females. Furthermore, pair bonding increased corticotropin-releasing hormone (CRH) expression in the bed nucleus of the stria terminalis (BNST) in male prairie voles, while defeat experience increased BNST CRH expression in females. Chemogenetic excitation of BNST CRH neurons during a short cohabitation with a new partner promoted a partner preference in stress-naïve prairie voles. Interestingly, chemogenetic inhibition of BNST CRH neurons during cohabitation with a new partner blocked partner preference in stress-naïve males but promoted preference in defeated males. Inhibition of BNST CRH neurons also blocked partner preference in stress-naïve females but did not alter preference behavior in defeated females. This study revealed sexual dimorphism in not only the impact of social defeat on pair bond formation, but also in the role BNST CRHergic neurons play in regulating changes in pair bonding following social conflict.
Q1

Absence of TAAR1 function increases methamphetamine-induced excitability of dorsal raphe serotonin neurons and drives binge-level methamphetamine intake
Rios S.M., Mootz J.R., Phillips T.J., Ingram S.L.
Abstract
Methamphetamine (MA) is a potent psychostimulant capable of exerting both rewarding and aversive effects, the balance of which likely drives variation in voluntary MA intake. Understanding the genetic factors underlying sensitivity to these effects of MA is critical for developing effective treatments. The activity of dorsal raphe serotonin neurons is linked to reward processing. Here, we performed whole-cell patch-clamp electrophysiology in dorsal raphe serotonin neurons from mice with high or low MA intake corresponding with high or low MA reward sensitivity. The MA drinking (MADR) mice consist of the MA reward sensitive MA high drinking (MAHDR) and the MA reward insensitive MA low drinking (MALDR) lines. MA is a trace amine-associated receptor 1 (TAAR1) agonist, and MAHDR mice are homozygous for a mutation in the Taar1 gene, Taar1
m1J
, that encodes non-functional TAAR1, whereas MALDR mice possess at least one copy of the reference Taar1
+
allele that encodes functional TAAR1. Our previous research using CRISPR-Cas9-generated MAHDR-Taar1
+/+
knock-in mice in which Taar1
m1J
was replaced with Taar1
+
, and non-edited MAHDR-Taar1
m1J/m1J
controls demonstrated that lack of TAAR1 function is critical for heightened MA consumption and MA reward sensitivity. Here, electrophysiological recordings in the MADR lines demonstrate a MA-induced decrease in dorsal raphe serotonin neuron activity from MALDR, but not MAHDR mice. However, in the presence of serotonin autoreceptor antagonists, MA potentiates dorsal raphe serotonin neuron activity of MAHDR, but not MALDR mice. Importantly, potentiation in the presence of the antagonists is abolished in knock-in mice expressing functional TAAR1. The knock-in mice did not display binge-level MA intake, consistent with the loss of MA-reward sensitivity previously reported in mice with functional TAAR1. Finally, because MA is a substrate of the serotonin transporter, we evaluated whether the serotonin transporter is necessary for MA-induced potentiation of dorsal raphe serotonin neuron activity in mice with non-functional TAAR1. The serotonin transporter antagonist fluoxetine blocks MA-induced potentiation for both MAHDR and MAHDR-Taar1
m1J/m1J
mice. Thus, TAAR1 function directly impacts MA reward sensitivity and MA intake and serves as a critical regulator of MA-induced activity of dorsal raphe serotonin neurons through its interaction with the serotonin transporter.
Q1

A molecular mechanism mediating clozapine-enhanced sensorimotor gating
Mantas I., Flais I., Branzell N., Ionescu T.M., Kim E., Zhang X., Cash D., Hengerer B., Svenningsson P.
Abstract
The atypical antipsychotic clozapine targets multiple receptor systems beyond the dopaminergic pathway and influences prepulse inhibition (PPI), a critical translational measure of sensorimotor gating. Since PPI is modulated by atypical antipsychotics such as risperidone and clozapine, we hypothesized that p11—an adaptor protein associated with anxiety- and depressive-like behaviors and G-protein-coupled receptor function—might modulate these effects. In this study, we assessed the role of p11 in clozapine’s PPI-enhancing effect by testing wild-type and global p11 knockout (KO) mice in response to haloperidol, risperidone, and clozapine. We also performed structural and functional brain imaging. Contrary to our expectation that anxiety-like p11-KO mice would exhibit an augmented startle response and heightened sensitivity to clozapine, PPI tests showed that p11-KO mice were unresponsive to the PPI-enhancing effects of risperidone and clozapine. Imaging revealed distinct regional brain volume differences and reduced hippocampal connectivity in p11-KO mice, with significantly blunted clozapine-induced connectivity changes in the CA1 region. Our findings highlight a novel role for p11 in modulating clozapine’s effects on sensorimotor gating and hippocampal connectivity, offering new insight into its functional pathways.
Q1

Brain and cardiovascular responses to acute stress in remitted and recurrent late-life depression
Kraynak T.E., Karim H.T., Banihashemi L., Krafty R.T., Butters M.A., Ajilore O.A., Taylor W.D., Andreescu C.
In individuals with remitted late-life depression (LLD), stress exposure can increase the likelihood of a new, recurrent depressive episode. Variability in the effect of stress on recurrence risk may reflect underlying brain and physiological processes mediating the stress response. We examined how subjective, physiological, and brain responses to an experimental stressor differs in older adults with and without remitted depression, and how these stress responses relate to future relapse. Participants were recruited through 3 sites and included 76 older adults with remitted LLD and 36 age-matched healthy comparison (HC) adults. Participants completed an acute stressor task during functional brain imaging with behavioral and cardiovascular monitoring. Remitted LLD participants were followed longitudinally to evaluate depression recurrence. Compared to HC, the remitted LLD group exhibited reduced stressor-evoked systolic blood pressure and heart rate responses, as well as reduced stressor-evoked posterior insula activity. This blunted stress response phenotype appeared more specific to the stable remitter group than the relapsing LLD group. Survival analyses demonstrated that greater stressor-evoked bed nucleus of the stria terminalis (BNST) activity was associated with faster time to recurrence. These findings add to a growing literature reporting so-called “blunted” stressor-evoked cardiovascular and brain reactivity in remitted depression. Moreover, they link the stress response in visceral interoceptive brain circuits with relapse vulnerability. Future work involving longer follow-up periods may reveal additional stress-related brain and behavioral predictors of recurrence in remitted LLD.
Q1

N-acetylcysteine for youth cannabis use disorder: randomized controlled trial main findings
Gray K.M., Tomko R.L., Baker N.L., McClure E.A., McRae-Clark A.L., Squeglia L.M.
Abstract
Cannabis use disorder is particularly prevalent and impairing among young people, and evidence-based treatments are limited. Prior trials of N-acetylcysteine, added to contingency management as a platform behavioral intervention, yielded positive findings in youth but not in adults. This trial sought to rigorously evaluate whether N-acetylcysteine is efficacious in youth when not paired with a robust behavioral treatment platform. Treatment-seeking youth with cannabis use disorder (N = 192, ages 14–21) were randomized to receive a double-blind 12-week course of oral N-acetylcysteine 1200 mg or placebo twice daily; all received weekly medical management and brief behavioral counseling. The primary efficacy outcome was the proportion of negative urine cannabinoid tests during treatment, compared between groups. An array of self-report and urine testing measures were examined secondarily to assess cannabis use reduction and cessation outcomes. The N-acetylcysteine and placebo groups did not differ in proportion of negative urine cannabinoid tests (RR = 0.93, 95% CI = 0.53, 1.64; p = 0.80) or self-reported cannabis abstinence (RR = 1.02, 95% CI = 0.63, 1.65; p = 0.93) during treatment. The mean percentage of cannabis use days and grams of cannabis used per using day decreased over time during treatment but did not differ between groups. More N-acetylcysteine than placebo treated participants reported gastrointestinal adverse events (63/98 versus 37/94, χ2
1 = 11.9 p < 0.001); adverse events were otherwise similar between groups. Findings indicate that N-acetylcysteine is not efficacious for youth cannabis use disorder when not paired with contingency management, highlighting the potentially crucial role of a robust behavioral treatment platform in facilitating prior positive efficacy findings with N-acetylcysteine.
Trial Registration: Clinicaltrials.gov identifier NCT03055377
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|
Frontiers Media S.A.
15 citations, 0.32%
|
|
Brill
13 citations, 0.27%
|
|
Duke University Press
11 citations, 0.23%
|
|
MIT Press
11 citations, 0.23%
|
|
Institute of Electrical and Electronics Engineers (IEEE)
11 citations, 0.23%
|
|
8 citations, 0.17%
|
|
Immanuel Kant Baltic Federal University
8 citations, 0.17%
|
|
The Pennsylvania State University Press
8 citations, 0.17%
|
|
Edinburgh University Press
7 citations, 0.15%
|
|
BMJ
7 citations, 0.15%
|
|
Japan Society of Civil Engineers
6 citations, 0.13%
|
|
University of Toronto Press Inc. (UTPress)
6 citations, 0.13%
|
|
Philosophy Documentation Center, Saint Louis University
5 citations, 0.11%
|
|
Hindawi Limited
5 citations, 0.11%
|
|
SciELO
5 citations, 0.11%
|
|
The Japanese Society of Clinical Cytology
5 citations, 0.11%
|
|
University of Bialystok
4 citations, 0.08%
|
|
University of Illinois Press
4 citations, 0.08%
|
|
Numdam (Numerisation de Documents Anciens Mathematiques)
4 citations, 0.08%
|
|
Association for Computing Machinery (ACM)
4 citations, 0.08%
|
|
University of Pittsburgh
4 citations, 0.08%
|
|
Academy of Management
4 citations, 0.08%
|
|
Surey Beatty & Sons
4 citations, 0.08%
|
|
Uniwersytet Jagiellonski - Wydawnictwo Uniwersytetu Jagiellonskiego
4 citations, 0.08%
|
|
Taras Shevchenko National University of Kyiv
3 citations, 0.06%
|
|
Architectural Institute of Japan
3 citations, 0.06%
|
|
Bibliopolis, Edizioni Di Filosofia E Scienze
3 citations, 0.06%
|
|
Scandinavian University Press / Universitetsforlaget AS
3 citations, 0.06%
|
|
IGI Global
3 citations, 0.06%
|
|
Hans Publishers
3 citations, 0.06%
|
|
Research Institute for Mechanics of National Research Lobachevsky State University of Nizhny Novgorod
3 citations, 0.06%
|
|
Ovid Technologies (Wolters Kluwer Health)
2 citations, 0.04%
|
|
Trans Tech Publications
2 citations, 0.04%
|
|
Japan Institute of Light Metals
2 citations, 0.04%
|
|
2 citations, 0.04%
|
|
The Philosophy Centre of the University of Lisbon
2 citations, 0.04%
|
|
American Veterinary Medical Association
2 citations, 0.04%
|
|
Vilnius University Press
2 citations, 0.04%
|
|
International Association for Plant Taxonomy
2 citations, 0.04%
|
|
S. Karger AG
2 citations, 0.04%
|
|
JSTOR
2 citations, 0.04%
|
|
AOSIS
2 citations, 0.04%
|
|
Equinox Publishing
2 citations, 0.04%
|
|
Pleiades Publishing
1 citation, 0.02%
|
|
1 citation, 0.02%
|
|
Proceedings of the National Academy of Sciences (PNAS)
1 citation, 0.02%
|
|
1 citation, 0.02%
|
|
Philosophy Documentation Center
1 citation, 0.02%
|
|
American Association for the Advancement of Science (AAAS)
1 citation, 0.02%
|
|
The Royal Society
1 citation, 0.02%
|
|
Universidade Federal do Rio Grande do Sul
1 citation, 0.02%
|
|
Public Library of Science (PLoS)
1 citation, 0.02%
|
|
Indiana University Press
1 citation, 0.02%
|
|
Universidad EAFIT
1 citation, 0.02%
|
|
1 citation, 0.02%
|
|
Lavoisier
1 citation, 0.02%
|
|
Universitat Salzburg
1 citation, 0.02%
|
|
Vilnius Gediminas Technical University
1 citation, 0.02%
|
|
Journal of the Society for the Advancement of Scandinavian Study
1 citation, 0.02%
|
|
Open Library of Humanities
1 citation, 0.02%
|
|
1 citation, 0.02%
|
|
1 citation, 0.02%
|
|
IOP Publishing
1 citation, 0.02%
|
|
1 citation, 0.02%
|
|
Pluto Journals
1 citation, 0.02%
|
|
Tohoku University Medical Press
1 citation, 0.02%
|
|
American Economic Association
1 citation, 0.02%
|
|
Pontificia Universidade Catolica de Sao Paulo
1 citation, 0.02%
|
|
Museu Paraense Emilio Goeldi
1 citation, 0.02%
|
|
Editions Ophrys
1 citation, 0.02%
|
|
Presses Universitaires Franc-Comtoises
1 citation, 0.02%
|
|
Institute of Philosophy, Russian Academy of Sciences
1 citation, 0.02%
|
|
Presses Universitaires du Septentrion
1 citation, 0.02%
|
|
American Public Health Association
1 citation, 0.02%
|
|
Samara State Technical University
1 citation, 0.02%
|
|
University Pub. Group
1 citation, 0.02%
|
|
University of Montreal
1 citation, 0.02%
|
|
NP Voprosy Ekonomiki
1 citation, 0.02%
|
|
Sirey
1 citation, 0.02%
|
|
Brazilian Administration Review
1 citation, 0.02%
|
|
Associacao Universitaria de Pesquisa em Psicopatologia Fundamental
1 citation, 0.02%
|
|
1 citation, 0.02%
|
|
Journal of Ayn Rand Studies Foundation
1 citation, 0.02%
|
|
Polish Academy of Arts and Sciences,Commission on the History of Science
1 citation, 0.02%
|
|
Show all (70 more) | |
100
200
300
400
500
600
700
|
Publishing organizations
50
100
150
200
250
300
|
|
University of Ottawa
278 publications, 4.83%
|
|
Queen's University at Kingston
149 publications, 2.59%
|
|
Université du Québec à Montréal
134 publications, 2.33%
|
|
University of Waterloo
120 publications, 2.09%
|
|
University of Guelph
118 publications, 2.05%
|
|
Université du Québec à Trois-Rivières
111 publications, 1.93%
|
|
York University
109 publications, 1.89%
|
|
University of Alberta
89 publications, 1.55%
|
|
McGill University
87 publications, 1.51%
|
|
University of Calgary
74 publications, 1.29%
|
|
Université Laval
68 publications, 1.18%
|
|
McMaster University
64 publications, 1.11%
|
|
Dalhousie University
59 publications, 1.03%
|
|
University of Saskatchewan
58 publications, 1.01%
|
|
Université de Sherbrooke
56 publications, 0.97%
|
|
Simon Fraser University
56 publications, 0.97%
|
|
Brock University
36 publications, 0.63%
|
|
University of Manitoba
36 publications, 0.63%
|
|
University of Victoria
34 publications, 0.59%
|
|
University of Toronto
34 publications, 0.59%
|
|
Wilfrid Laurier University
33 publications, 0.57%
|
|
Trent University
32 publications, 0.56%
|
|
Toronto Metropolitan University
27 publications, 0.47%
|
|
University of Windsor
27 publications, 0.47%
|
|
Laurentian University
25 publications, 0.43%
|
|
Ege University
23 publications, 0.4%
|
|
Carleton University
23 publications, 0.4%
|
|
University of Lethbridge
23 publications, 0.4%
|
|
University of Liège
19 publications, 0.33%
|
|
University of Regina
18 publications, 0.31%
|
|
Vanderbilt University
14 publications, 0.24%
|
|
Lakehead University
14 publications, 0.24%
|
|
Fordham University
12 publications, 0.21%
|
|
Princeton University
11 publications, 0.19%
|
|
Paris Cité University
11 publications, 0.19%
|
|
Sorbonne University
10 publications, 0.17%
|
|
University of Sydney
10 publications, 0.17%
|
|
University of Chicago
10 publications, 0.17%
|
|
University of Texas at Austin
10 publications, 0.17%
|
|
Université Catholique de Louvain
9 publications, 0.16%
|
|
Cornell University
9 publications, 0.16%
|
|
Pennsylvania State University
9 publications, 0.16%
|
|
University of Cambridge
8 publications, 0.14%
|
|
Western University
8 publications, 0.14%
|
|
Institute for Research in Fundamental Sciences
7 publications, 0.12%
|
|
University of Auckland
7 publications, 0.12%
|
|
University at Buffalo, State University of New York
7 publications, 0.12%
|
|
University of Illinois Urbana-Champaign
7 publications, 0.12%
|
|
University of Kentucky
7 publications, 0.12%
|
|
University of Lorraine
6 publications, 0.1%
|
|
University of Geneva
6 publications, 0.1%
|
|
Johns Hopkins University
6 publications, 0.1%
|
|
Monash University
6 publications, 0.1%
|
|
Sungkyunkwan University
6 publications, 0.1%
|
|
Rutgers, The State University of New Jersey
6 publications, 0.1%
|
|
University of California, Irvine
6 publications, 0.1%
|
|
University of California, Santa Barbara
6 publications, 0.1%
|
|
Purdue University
6 publications, 0.1%
|
|
Brown University
6 publications, 0.1%
|
|
East Carolina University
6 publications, 0.1%
|
|
Brandon University
6 publications, 0.1%
|
|
University of Louisville
6 publications, 0.1%
|
|
University of Colorado Boulder
6 publications, 0.1%
|
|
Université de Moncton
6 publications, 0.1%
|
|
University of Prince Edward Island
6 publications, 0.1%
|
|
Tel Aviv University
5 publications, 0.09%
|
|
Katholieke Universiteit Leuven
5 publications, 0.09%
|
|
Aix-Marseille University
5 publications, 0.09%
|
|
Wuhan University
5 publications, 0.09%
|
|
University of Neuchâtel
5 publications, 0.09%
|
|
University of Oxford
5 publications, 0.09%
|
|
King's College London
5 publications, 0.09%
|
|
Yale University
5 publications, 0.09%
|
|
Universite Libre de Bruxelles
5 publications, 0.09%
|
|
Victoria University of Wellington
5 publications, 0.09%
|
|
Columbia University
5 publications, 0.09%
|
|
Auburn University
5 publications, 0.09%
|
|
North Carolina State University
5 publications, 0.09%
|
|
Northwestern University
5 publications, 0.09%
|
|
Harvard University
5 publications, 0.09%
|
|
University of California, Davis
5 publications, 0.09%
|
|
University of California, Riverside
5 publications, 0.09%
|
|
Northeastern University
5 publications, 0.09%
|
|
Western Michigan University
5 publications, 0.09%
|
|
St. Francis Xavier University
5 publications, 0.09%
|
|
University of Wisconsin–Madison
5 publications, 0.09%
|
|
University of Cincinnati
5 publications, 0.09%
|
|
Université du Québec à Rimouski
5 publications, 0.09%
|
|
University of Helsinki
4 publications, 0.07%
|
|
University of Fribourg
4 publications, 0.07%
|
|
University of Birmingham
4 publications, 0.07%
|
|
University of Newcastle Australia
4 publications, 0.07%
|
|
Stanford University
4 publications, 0.07%
|
|
Boston University
4 publications, 0.07%
|
|
Catholic University of America
4 publications, 0.07%
|
|
Duke University
4 publications, 0.07%
|
|
Syracuse University
4 publications, 0.07%
|
|
San Francisco State University
4 publications, 0.07%
|
|
University of Arizona
4 publications, 0.07%
|
|
Northern Illinois University
4 publications, 0.07%
|
|
Show all (70 more) | |
50
100
150
200
250
300
|
Publishing organizations in 5 years
1
2
3
4
5
6
7
8
|
|
York University
8 publications, 4.62%
|
|
University of Toronto
6 publications, 3.47%
|
|
McGill University
5 publications, 2.89%
|
|
Université du Québec à Montréal
4 publications, 2.31%
|
|
Institute for Research in Fundamental Sciences
3 publications, 1.73%
|
|
University of Lorraine
3 publications, 1.73%
|
|
Sorbonne University
3 publications, 1.73%
|
|
University of Alberta
3 publications, 1.73%
|
|
University of Ottawa
3 publications, 1.73%
|
|
Aix-Marseille University
2 publications, 1.16%
|
|
University of Zurich
2 publications, 1.16%
|
|
École normale supérieure Paris-Saclay
2 publications, 1.16%
|
|
Manchester Metropolitan University
2 publications, 1.16%
|
|
Universite Libre de Bruxelles
2 publications, 1.16%
|
|
University of Melbourne
2 publications, 1.16%
|
|
St. Francis Xavier University
2 publications, 1.16%
|
|
McMaster University
2 publications, 1.16%
|
|
Sorbonne Paris Nord University
2 publications, 1.16%
|
|
Western University
2 publications, 1.16%
|
|
Université Laval
2 publications, 1.16%
|
|
Brock University
2 publications, 1.16%
|
|
Quinnipiac University
2 publications, 1.16%
|
|
University of Tyumen
1 publication, 0.58%
|
|
Bilkent University
1 publication, 0.58%
|
|
University of Qom
1 publication, 0.58%
|
|
Tel Aviv University
1 publication, 0.58%
|
|
Dalian University of Technology
1 publication, 0.58%
|
|
Katholieke Universiteit Leuven
1 publication, 0.58%
|
|
University of Lisbon
1 publication, 0.58%
|
|
University of Strasbourg
1 publication, 0.58%
|
|
Bordeaux Montaigne University
1 publication, 0.58%
|
|
University of Bordeaux
1 publication, 0.58%
|
|
University of Helsinki
1 publication, 0.58%
|
|
University of Gothenburg
1 publication, 0.58%
|
|
Wuhan University
1 publication, 0.58%
|
|
University of Lausanne
1 publication, 0.58%
|
|
University of Neuchâtel
1 publication, 0.58%
|
|
University of Oxford
1 publication, 0.58%
|
|
Aarhus University
1 publication, 0.58%
|
|
King's College London
1 publication, 0.58%
|
|
Kanazawa University
1 publication, 0.58%
|
|
University of Sydney
1 publication, 0.58%
|
|
University of Pavia
1 publication, 0.58%
|
|
Pennsylvania State University
1 publication, 0.58%
|
|
Iowa State University
1 publication, 0.58%
|
|
University of Otago
1 publication, 0.58%
|
|
Macquarie University
1 publication, 0.58%
|
|
Sungkyunkwan University
1 publication, 0.58%
|
|
Ulsan National Institute of Science and Technology
1 publication, 0.58%
|
|
North Carolina State University
1 publication, 0.58%
|
|
Oregon State University
1 publication, 0.58%
|
|
New York University
1 publication, 0.58%
|
|
University of Chicago
1 publication, 0.58%
|
|
National and Kapodistrian University of Athens
1 publication, 0.58%
|
|
National University of La Plata
1 publication, 0.58%
|
|
University of Illinois Urbana-Champaign
1 publication, 0.58%
|
|
Kwantlen Polytechnic University
1 publication, 0.58%
|
|
Queen's University at Kingston
1 publication, 0.58%
|
|
Leiden University
1 publication, 0.58%
|
|
Indiana University–Purdue University Indianapolis
1 publication, 0.58%
|
|
University of Amsterdam
1 publication, 0.58%
|
|
Bucknell University
1 publication, 0.58%
|
|
Laurentian University
1 publication, 0.58%
|
|
Carleton University
1 publication, 0.58%
|
|
University of Saskatchewan
1 publication, 0.58%
|
|
University of Guelph
1 publication, 0.58%
|
|
University of Miami
1 publication, 0.58%
|
|
Western Kentucky University
1 publication, 0.58%
|
|
University of Kentucky
1 publication, 0.58%
|
|
Indiana University Bloomington
1 publication, 0.58%
|
|
Université Clermont Auvergne
1 publication, 0.58%
|
|
Paris Sciences et Lettres
1 publication, 0.58%
|
|
University of Memphis
1 publication, 0.58%
|
|
Portland State University
1 publication, 0.58%
|
|
Show all (44 more) | |
1
2
3
4
5
6
7
8
|
Publishing countries
500
1000
1500
2000
2500
3000
|
|
Canada
|
Canada, 2790, 48.49%
Canada
2790 publications, 48.49%
|
USA
|
USA, 763, 13.26%
USA
763 publications, 13.26%
|
France
|
France, 159, 2.76%
France
159 publications, 2.76%
|
United Kingdom
|
United Kingdom, 67, 1.16%
United Kingdom
67 publications, 1.16%
|
Belgium
|
Belgium, 46, 0.8%
Belgium
46 publications, 0.8%
|
Australia
|
Australia, 34, 0.59%
Australia
34 publications, 0.59%
|
Turkey
|
Turkey, 30, 0.52%
Turkey
30 publications, 0.52%
|
Republic of Korea
|
Republic of Korea, 24, 0.42%
Republic of Korea
24 publications, 0.42%
|
Germany
|
Germany, 22, 0.38%
Germany
22 publications, 0.38%
|
Switzerland
|
Switzerland, 20, 0.35%
Switzerland
20 publications, 0.35%
|
New Zealand
|
New Zealand, 18, 0.31%
New Zealand
18 publications, 0.31%
|
China
|
China, 13, 0.23%
China
13 publications, 0.23%
|
Brazil
|
Brazil, 13, 0.23%
Brazil
13 publications, 0.23%
|
South Africa
|
South Africa, 12, 0.21%
South Africa
12 publications, 0.21%
|
Israel
|
Israel, 11, 0.19%
Israel
11 publications, 0.19%
|
Iran
|
Iran, 10, 0.17%
Iran
10 publications, 0.17%
|
Denmark
|
Denmark, 9, 0.16%
Denmark
9 publications, 0.16%
|
Netherlands
|
Netherlands, 9, 0.16%
Netherlands
9 publications, 0.16%
|
Norway
|
Norway, 8, 0.14%
Norway
8 publications, 0.14%
|
Italy
|
Italy, 7, 0.12%
Italy
7 publications, 0.12%
|
Finland
|
Finland, 7, 0.12%
Finland
7 publications, 0.12%
|
Ireland
|
Ireland, 6, 0.1%
Ireland
6 publications, 0.1%
|
Argentina
|
Argentina, 5, 0.09%
Argentina
5 publications, 0.09%
|
Greece
|
Greece, 5, 0.09%
Greece
5 publications, 0.09%
|
India
|
India, 5, 0.09%
India
5 publications, 0.09%
|
Spain
|
Spain, 4, 0.07%
Spain
4 publications, 0.07%
|
Sweden
|
Sweden, 4, 0.07%
Sweden
4 publications, 0.07%
|
Japan
|
Japan, 4, 0.07%
Japan
4 publications, 0.07%
|
Lebanon
|
Lebanon, 3, 0.05%
Lebanon
3 publications, 0.05%
|
Luxembourg
|
Luxembourg, 3, 0.05%
Luxembourg
3 publications, 0.05%
|
Uganda
|
Uganda, 3, 0.05%
Uganda
3 publications, 0.05%
|
Montenegro
|
Montenegro, 3, 0.05%
Montenegro
3 publications, 0.05%
|
Russia
|
Russia, 2, 0.03%
Russia
2 publications, 0.03%
|
Ghana
|
Ghana, 2, 0.03%
Ghana
2 publications, 0.03%
|
Mexico
|
Mexico, 2, 0.03%
Mexico
2 publications, 0.03%
|
UAE
|
UAE, 2, 0.03%
UAE
2 publications, 0.03%
|
Romania
|
Romania, 2, 0.03%
Romania
2 publications, 0.03%
|
Portugal
|
Portugal, 1, 0.02%
Portugal
1 publication, 0.02%
|
Vietnam
|
Vietnam, 1, 0.02%
Vietnam
1 publication, 0.02%
|
Iraq
|
Iraq, 1, 0.02%
Iraq
1 publication, 0.02%
|
Iceland
|
Iceland, 1, 0.02%
Iceland
1 publication, 0.02%
|
Czech Republic
|
Czech Republic, 1, 0.02%
Czech Republic
1 publication, 0.02%
|
Ecuador
|
Ecuador, 1, 0.02%
Ecuador
1 publication, 0.02%
|
Show all (13 more) | |
500
1000
1500
2000
2500
3000
|
Publishing countries in 5 years
10
20
30
40
50
60
|
|
Canada
|
Canada, 52, 30.06%
Canada
52 publications, 30.06%
|
France
|
France, 26, 15.03%
France
26 publications, 15.03%
|
USA
|
USA, 26, 15.03%
USA
26 publications, 15.03%
|
Australia
|
Australia, 4, 2.31%
Australia
4 publications, 2.31%
|
United Kingdom
|
United Kingdom, 4, 2.31%
United Kingdom
4 publications, 2.31%
|
Iran
|
Iran, 4, 2.31%
Iran
4 publications, 2.31%
|
Switzerland
|
Switzerland, 4, 2.31%
Switzerland
4 publications, 2.31%
|
Belgium
|
Belgium, 3, 1.73%
Belgium
3 publications, 1.73%
|
China
|
China, 2, 1.16%
China
2 publications, 1.16%
|
Argentina
|
Argentina, 2, 1.16%
Argentina
2 publications, 1.16%
|
Ireland
|
Ireland, 2, 1.16%
Ireland
2 publications, 1.16%
|
Netherlands
|
Netherlands, 2, 1.16%
Netherlands
2 publications, 1.16%
|
Republic of Korea
|
Republic of Korea, 2, 1.16%
Republic of Korea
2 publications, 1.16%
|
Romania
|
Romania, 2, 1.16%
Romania
2 publications, 1.16%
|
Russia
|
Russia, 1, 0.58%
Russia
1 publication, 0.58%
|
Portugal
|
Portugal, 1, 0.58%
Portugal
1 publication, 0.58%
|
Greece
|
Greece, 1, 0.58%
Greece
1 publication, 0.58%
|
Denmark
|
Denmark, 1, 0.58%
Denmark
1 publication, 0.58%
|
Israel
|
Israel, 1, 0.58%
Israel
1 publication, 0.58%
|
Iraq
|
Iraq, 1, 0.58%
Iraq
1 publication, 0.58%
|
Italy
|
Italy, 1, 0.58%
Italy
1 publication, 0.58%
|
New Zealand
|
New Zealand, 1, 0.58%
New Zealand
1 publication, 0.58%
|
Finland
|
Finland, 1, 0.58%
Finland
1 publication, 0.58%
|
Sweden
|
Sweden, 1, 0.58%
Sweden
1 publication, 0.58%
|
South Africa
|
South Africa, 1, 0.58%
South Africa
1 publication, 0.58%
|
Japan
|
Japan, 1, 0.58%
Japan
1 publication, 0.58%
|
10
20
30
40
50
60
|